@article{oai:az.repo.nii.ac.jp:00004845,
 author = {菅沼, 常徳 and 宇根, 有美 and 山田, 隆紹 and Suganuma, Tsunenori and Une, Yumi and Yamada, Takatsugu},
 journal = {麻布大学雑誌, Journal of Azabu University},
 month = {Mar},
 note = {骨病変を発症する因子を保有しているサルを飼育温度が高い施設から飼育温度が低い施設に移動したところ,10匹中6匹が発症した。再現された病変は過去の症例と同じであった。これらのことから,リスザルの過骨症の発症には,低温が深く関与していることがわかった。骨の増生は,下顎骨と長管骨すなわち尺骨,脛骨,尾骨血管弓で早期にかつ高度の病変がみられ,漸次進行していった。進行例には骨粗鬆症様の病変が観察された。なお,プロスタグランデインE2,CaおよびPにおいても重症例と対照例とで有意差は認められなかった。, The squirrel monkeys with the factor having a tendency to get bone lesions were moved from the facility where the temperature was high to a low facility. Then, bone lesions occurred in six of ten monkeys. The reproduced lesions is corresponding to our previous cases. From the above-mentioned, it has been understood that the low temperature closely related to the appearance of squirrel monkey's hyperostosis. Hyperostosis appears in the mandibule and long bonesof the extremities (ex. ulna, tibia , hemal arches of tailbone) at the early stage, and the lesions had progressed gradually. The osteoporosis ミ like lesions were observed in advanced cases. In addition, as for the value of Prostagrandin E 2, serum calcium, and phosphorus, a significant difference was not admitted by the advanced cases and control., P(論文), 特集, application/pdf, FEATURE ARTICLES},
 pages = {137--139},
 title = {リスザルの皮質過骨症の経時的観察},
 volume = {11/12},
 year = {2006},
 yomi = {スガヌマ, ツネノリ and ウネ, ユミ and ヤマダ, タカツグ}
}