{"created":"2023-06-19T07:19:01.127578+00:00","id":4686,"links":{},"metadata":{"_buckets":{"deposit":"0ac3a2cb-55f9-4be1-b38e-ff2538d8f1f5"},"_deposit":{"created_by":4,"id":"4686","owners":[4],"pid":{"revision_id":0,"type":"depid","value":"4686"},"status":"published"},"_oai":{"id":"oai:az.repo.nii.ac.jp:00004686","sets":["17:251:307","17:253:322","17:257:353","17:37:217","17:38:265","17:40:278","17:40:287","31:181:490"]},"author_link":["20413","20420","20415","20412","20399","20400","20407","20411","20419","20404","20417","20410","20414","20402","20418","20409","20416","20408","20406","20405","20403","20401"],"item_12_biblio_info_16":{"attribute_name":"書誌情報","attribute_value_mlt":[{"bibliographicIssueDates":{"bibliographicIssueDate":"2005-03-31","bibliographicIssueDateType":"Issued"},"bibliographicPageEnd":"231","bibliographicPageStart":"229","bibliographicVolumeNumber":"9/10","bibliographic_titles":[{"bibliographic_title":"麻布大学雑誌"},{"bibliographic_title":"Journal of Azabu University","bibliographic_titleLang":"en"}]}]},"item_12_description_13":{"attribute_name":"抄録","attribute_value_mlt":[{"subitem_description":"ヒトGSK3β遺伝子のプロモーター領域について多型解析を行った結果,統合失調症および躁鬱病と有意に相関する遺伝子多型は認められなかった。このことは,mRNAの安定性に関与する領域,トランス調節因子などについて解析する必要があることを示唆している。","subitem_description_type":"Abstract"}]},"item_12_description_14":{"attribute_name":"Abstract","attribute_value_mlt":[{"subitem_description":"The aim of this study is to reveal polymorphism in genes associated with psychiatric disorders and neurodegenerative diseases. Genomic DNA from patients affected by schizophrenia or bipolar, and normal humans were analyzed. Comparing the psychiatric disorders with normal humans, no significant differences in a ratio of polymorphism found throughout the regulatory region of GSK3β gene (promoter and 5'-UTR). Lithium, a therapeutic agent for psychiatric disorder such as bipolar, inhibits GSK3βand production of amyloidβ, a main component of senile plaques in Alzheimer's disease. In addition, the level of GSK3βis decreased in schizophrenia patients and increased depending on aging. The results in this study lead us to consider that 3'-UTR of GSK3β gene and trans acting factors for GSK3β transcription should be studied. In conclusion, the present data may provide us basic and important information that promote revealing the molecular mechanism for regulation of GSK3βgene expression.","subitem_description_type":"Other"}]},"item_12_description_2":{"attribute_name":"ページ属性","attribute_value_mlt":[{"subitem_description":"P(論文)","subitem_description_type":"Other"}]},"item_12_description_3":{"attribute_name":"記事種別","attribute_value_mlt":[{"subitem_description":"特集","subitem_description_type":"Other"}]},"item_12_description_34":{"attribute_name":"format","attribute_value_mlt":[{"subitem_description":"application/pdf","subitem_description_type":"Other"}]},"item_12_description_4":{"attribute_name":"Type","attribute_value_mlt":[{"subitem_description":"FEATURE ARTICLES","subitem_description_type":"Other"}]},"item_12_publisher_19":{"attribute_name":"出版者","attribute_value_mlt":[{"subitem_publisher":"麻布大学"}]},"item_12_publisher_20":{"attribute_name":"Publisher","attribute_value_mlt":[{"subitem_publisher":"Azabu 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